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Deformation mechanism of ω-enriched Ti–Nb-based gum metal: Dislocation channeling and deformation induced ω–β transformation

[Display omitted] Gum metal, a class of multifunctional β titanium alloys, has attracted much attention in the past decade due to its initially-proposed dislocation–free deformation mechanism based on giant faults, i.e., macroscopic planar defects carrying significant plastic strain. Special deforma...

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Bibliographic Details
Published in:Acta materialia 2015-11, Vol.100, p.290-300
Main Authors: Lai, M.J., Tasan, C.C., Raabe, D.
Format: Article
Language:English
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Summary:[Display omitted] Gum metal, a class of multifunctional β titanium alloys, has attracted much attention in the past decade due to its initially-proposed dislocation–free deformation mechanism based on giant faults, i.e., macroscopic planar defects carrying significant plastic strain. Special deformation features were observed in these alloys, such as plastic flow localization, pronounced surface steps, low work hardening, and large elongation. These were all proposed to arise from the special giant fault mechanism activated in the β-Ti matrix, while the initial presence or mechanically-induced formation of other phases was debated in several follow-up studies. Here, we set off with Ti–Nb-based gum metal samples with confirmed presence of large amounts of nanometer-sized hexagonal ω particles. Deformation experiments demonstrate all the features observed in the original reports, mentioned above. However, careful characterization reveals that the deformation bands (similar to giant faults) where plastic flow localized are “dislocation channels” that are depleted of ω phase. These channels are proposed to form by a {112}〈111〉 dislocation dissociation mechanism, promoting reverse transformation of the ω phase into the β phase. The deformation induced ω–β transformation and the associated dislocation channeling process can explain the presence of the aforementioned special deformation features in the current gum metal.
ISSN:1359-6454
1873-2453
DOI:10.1016/j.actamat.2015.08.047