Maize w3 disrupts homogentisate solanesyl transferase (ZmHst) and reveals a plastoquinone‐9 independent path for phytoene desaturation and tocopherol accumulation in kernels

Summary Maize white seedling 3 (w3) has been used to study carotenoid deficiency for almost 100 years, although the molecular basis of the mutation has remained unknown. Here we show that the w3 phenotype is caused by disruption of the maize gene for homogentisate solanesyl transferase (HST), which...

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Bibliographic Details
Published in:	The Plant journal : for cell and molecular biology 2018-03, Vol.93 (5), p.799-813
Main Authors:	Hunter, Charles T., Saunders, Jonathan W., Magallanes‐Lundback, Maria, Christensen, Shawn A., Willett, Denis, Stinard, Philip S., Li, Qin‐Bao, Lee, Kwanghee, DellaPenna, Dean, Koch, Karen E.
Format:	Article
Language:	English
Subjects:	Abscisic acid Accumulation albino Biosynthesis Carotenoids color Corn Desaturase Desaturation Electron transfer Embryos Endosperm genes Grain homogentisate solanesyl transferase homozygosity Kernels leaves mutants mutation nutrient deficiencies oxidants Oxidative stress Oxidizing agents Phenotypes Photosynthesis phytoene phytoene desaturase Plant tissues plastoquinone seed maturation Seedlings tissues tocochromanols Tocopherol Tocopherols tocotrienols transferases Vitamin E white seedling 3 Zea mays
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