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Influence of cyclic fatigue in water on the load-bearing capacity of dental bridges made of zirconia

The humid atmosphere and permanent occurrence of chewing forces in the oral environment lead to degradation of ceramics used for prosthetic restorations. The aim of this in vitro study was to evaluate the influence of artificial aging on the load-bearing capacity of four-unit bridges, with both unda...

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Bibliographic Details
Published in:Acta biomaterialia 2008-09, Vol.4 (5), p.1440-1447
Main Authors: Kohorst, Philipp, Dittmer, Marc Philipp, Borchers, Lothar, Stiesch-Scholz, Meike
Format: Article
Language:English
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Summary:The humid atmosphere and permanent occurrence of chewing forces in the oral environment lead to degradation of ceramics used for prosthetic restorations. The aim of this in vitro study was to evaluate the influence of artificial aging on the load-bearing capacity of four-unit bridges, with both undamaged and predamaged zirconia frameworks. Additionally, different parameters for chewing simulation have been investigated and a finite element analysis was made to predict the location of highest tensile stresses within the bridges. A total of 60 frameworks were milled from presintered zirconia and divided into six homogeneous groups. Prior to veneering, frameworks of two groups were “damaged” by a defined saw cut similar to an accidental flaw generated during shape cutting. After veneering, FPDs were subjected to thermal and mechanical cycling – with the exception of control groups. The load-bearing capacity of tested FPDs was significantly reduced by artificial aging. In comparison to unaged specimens, fracture resistance decreased by about 40%, whereas preliminary damage did not have a significant effect. Increasing number of cycles and increasing upper load limit failed to show any additional effect on fracture force. To predict the progression of degradation under the terms of in vitro simulation for even longer periods, further aging experiments are required.
ISSN:1742-7061
1878-7568
DOI:10.1016/j.actbio.2008.04.012