Influences of Potential for Hostility, Type A Behavior, and Parental History of Hypertension on Adolescents' Cardiovascular Responses During Stress

ABSTRACT We investigated the influences of Type A behavior, potential for hostility, and parental history of diagnosed hypertension on adolescents’ cardiovascular responses to three behavioral stressors. The participants were 72 male and 99 female adolescents enrolled in grades 6 to 12. Results show...

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Bibliographic Details
Published in:Psychophysiology 1988-09, Vol.25 (5), p.503-511
Main Authors: McCann, Barbara S., Matthews, Karen A.
Format: Article
Language:English
Subjects:
Adolescent
Adolescents
Arousal - physiology
Biological and medical sciences
Blood Pressure
Family history
Female
Fundamental and applied biological sciences. Psychology
Heart Rate
Hostility
Humans
Hypertension
Hypertension - genetics
Hypertension - physiopathology
Illness and personality
Male
Psychology and medicine
Psychology. Psychoanalysis. Psychiatry
Psychology. Psychophysiology
Risk Factors
Stress, Psychological - complications
Type A
Type A Personality
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Summary:ABSTRACT We investigated the influences of Type A behavior, potential for hostility, and parental history of diagnosed hypertension on adolescents’ cardiovascular responses to three behavioral stressors. The participants were 72 male and 99 female adolescents enrolled in grades 6 to 12. Results showed that adolescents with a hypertensive parent had larger diastolic blood pressure responses during all three stressors than did adolescents without hypertensive parents; this effect was particularly pronounced among the Type As. Adolescents rated as high on potential for hostility had elevated systolic and diastolic blood pressure responses, particularly during isometric exercise. Results regarding parental history of hypertension and Type A replicate previous studies in adolescents. Results regarding potential for hostility showed for the first time that early signs of hostility are related to psychophysiological responses thought to be pathophysiological mechanisms in the etiology of cardiovascular diseases.
ISSN:0048-5772
1469-8986
1540-5958
DOI:10.1111/j.1469-8986.1988.tb01885.x